Team:St Andrews

From 2011.igem.org

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Revision as of 14:27, 15 July 2011

Description
Project Diary
Safety Form

The Kill Switch
Modelling
Parts Submitted

Drug Delivery
Biosafety
Conjugation

"Is iGEM Fair?"
Panel Debate
Reflective Essays

The Team
Sponsors
Collaboration

Project Description

Antimicrobial Peptides have been an area of great interest for many years. They form part of an evolutionarily conserved innate immune response.

For the 2011 St Andrews iGEM project we are creating a ‘kill switch’ where we will be inserting a gene (protogrin-1) coding for Anti-Microbial Peptides (AMP’s) into E. Coli. AMP’s cause the death of bacterial cells by displacement of lipids and alteration of the membrane structure. The idea is to then use different promoters to switch on the expression of these proteins in certain conditions (we envisage that this would have many practical applications, some of which we hope to maybe explore later on).

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+|style=font-size:1em;| Antimicrobial Peptides have been an area of great interest for many years.  They form part of an evolutionarily conserved innate immune response.
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+|style=font-size:1em;|For the 2011 St Andrews iGEM project we are creating a ‘kill switch’ where we will be inserting a gene (protogrin-1) coding for Anti-Microbial Peptides (AMP’s) into E. Coli.   AMP’s cause the death of bacterial cells by displacement of lipids and alteration of the membrane structure.  The idea is to then use different promoters to switch on the expression of these proteins in certain conditions (we envisage that this would have many practical applications, some of which we hope to maybe explore later on).
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-|style=font-size:1em;| Antimicrobial Peptides have been an area of great interest for many years.  They form part of an evolutionarily conserved innate immune response.
-|-
-|style=font-size:1em;|For the 2011 St Andrews iGEM project we are creating a ‘kill switch’ where we will be inserting a gene (protogrin-1) coding for Anti-Microbial Peptides (AMP’s) into E. Coli.   AMP’s cause the death of bacterial cells by displacement of lipids and alteration of the membrane structure.  The idea is to then use different promoters to switch on the expression of these proteins in certain conditions (we envisage that this would have many practical applications, some of which we hope to maybe explore later on).